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| Year : 2013 | Volume
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| Issue : 5 | Page : 1023 |
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| Authors reply |
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Mohammad Taghi Goodarzi
Research Center for Molecular Medicine, Hamadan University of Medical Sciences, 65178 Hamadan, Iran
Click here for correspondence address and email
| Date of Web Publication | 12-Sep-2013 |
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How to cite this article:
Goodarzi MT. Authors reply. Saudi J Kidney Dis Transpl 2013;24:1023 |
To the Editor,
On behalf of all the authors, I thank Dr. Roomizadeh and his colleague for their interest in our article and their comments. Here, I will try to explain some points:
- In our article, the citrate excretion (mg/g creatinine), urinary citrate concentration (mg/L) and daily citrate excretion (mg/24 h) were lower in stone formers compared with that in healthy subjects [Table 3], which is quite different from what the authors of the letters had stated. Furthermore, in our abstract, we concluded that the "prevalence" of hypocitraturia in stone formers was higher than those of the healthy group, which is exactly correct and is consistent with our findings and other published reports.
- Our finding was similar to other previously published reports. However, as the letter's authors indicated, there are still some reports that show no difference in citrate excretion between stone formers and healthy subjects.
- The case group in our study was stone formers as shown by the title; therefore, our sample was not restricted to calcium stone formers. However, we agree that if we only selected calcium stone formers, the conclusion would have been more precise.
- In our article, we pointed out the limitations of our study including the small number of the study population.
Correspondence Address:
Mohammad Taghi Goodarzi
Research Center for Molecular Medicine, Hamadan University of Medical Sciences, 65178 Hamadan
Iran
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DOI: 10.4103/1319-2442.118084 
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