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 Indian J Med Microbiol  
 

Figure 3: Representative photomicrographs of histopathological changes of the kidney (A-E) and liver (F-J) after renal I/R in rats (hematoxylin & eosin, ×40). (A and F) Kidney and liver sections, respectively, from sham-operated group demonstrating the normal architecture of both organs. (B and G) Kidney and liver sections, respectively, from the vehicle-pretreated I/R group showing tubular dilatation (a plus sign), cast formation (a double-headed arrow), tubular cell necrosis (a single-headed arrow), and leukocyte infiltration (double arrows) in the kidney (B); irregularity of hepatic cell cords, leukocyte infiltration (double arrows), and areas of necrosis of hepatocytes (a star) in the liver (G). Kidney (C-E) and liver (H-J) sections from FEB + I/R, ALA + I/R, and (FEB + ALA) + I/R groups showing less histopathological alterations with overall preservation of normal organ architecture.

Figure 3: Representative photomicrographs of histopathological changes of the kidney (A-E) and liver (F-J) after renal I/R in rats (hematoxylin & eosin, ×40). (A and F) Kidney and liver sections, respectively, from sham-operated group demonstrating the normal architecture of both organs. (B and G) Kidney and liver sections, respectively, from the vehicle-pretreated I/R group showing tubular dilatation (a plus sign), cast formation (a double-headed arrow), tubular cell necrosis (a single-headed arrow), and leukocyte infiltration (double arrows) in the kidney (B); irregularity of hepatic cell cords, leukocyte infiltration (double arrows), and areas of necrosis of hepatocytes (a star) in the liver (G). Kidney (C-E) and liver (H-J) sections from FEB + I/R, ALA + I/R, and (FEB + ALA) + I/R groups showing less histopathological alterations with overall preservation of normal organ architecture.